Fahd al-Quso Dead: Airstrike Kills Senior Al-Qaida Leader In Yemen

SANAA, Yemen -- An airstrike Sunday killed a top al-Qaida leader on the FBI's most wanted list for his role in the 2000 bombing of the USS Cole warship, Yemeni officials said. The airstrike resembled earlier U.S. drone attacks, but the U.S. did not immediately confirm it.

Fahd al-Quso was hit by a missile as he stepped out of his vehicle, along with another al-Qaida operative in the southern Shabwa province, Yemeni military officials said. They were speaking on condition of anonymity in accordance with military regulations.

The airstrike came as the U.S. and Yemen cooperate in a battle against al-Qaida in southern Yemen.

Al-Quso, 37, was on the FBI's most wanted list, with a $5 million reward for information leading to his capture. He was indicted in the U.S. for his role in the 2000 bombing in the USS Cole in Aden, Yemen, in which 17 American sailors were killed and 39 injured.

He served more than five years in a Yemeni prison for his role in the attack and was released in 2007. He briefly escaped prison in 2003 but later turned himself in to serve the rest of his sentence.

A telephone text message claiming to be from al-Qaida media arm confirmed al-Quso was killed in the strike.

Al-Quso was also one of the most senior al-Qaida leaders publicly linked to the 2009 Christmas airliner attack. He has allegedly met with the suspected bomber Umar Farouk Abdulmutallab in Yemen before he left on his way to execute his failed bombing over Detroit.

In December 2010, al-Quso was designated a global terrorist by the State Department, an indication that his role in al-Qaida's Yemen branch had grown more prominent.

Local Yemeni official Abu Bakr bin Farid and the Yemeni Embassy in Washington confirmed al-Quso was killed in Rafd, a remote mountain valley in Shabwa. It is the area where many of al-Qaida leaders are believed to have taken cover, including the U.S.-born cleric Anwar al-Awlaki, killed in a U.S. airstrike in Yemen last year.

Yemeni government officials reported that Al-Quso and al-Awlaki were killed in an airstrike in 2009 in Rafd, but they both resurfaced alive.

Al-Quso was known for his ability to move in disguise. He was from the same tribe as al-Awlaki, and local tribesmen said he was a close aide. He studied ultraconservative Salafi Islam as a teenager in northern Yemen, then returned home to learn welding.

The White House and the State Department had no immediate comment.

Yemen's government has been waging an offensive on al-Qaida militants, who have taken advantage of the country's political turmoil over the last year to expand their hold in the south.

The new Yemeni president has promised improved cooperation with the U.S. to combat the militants. On Saturday, he said the fight against al-Qaida is in its early stages.

Al-Quso's association with al-Qaida dated back more than a decade, when he met with Osama bin Laden in Afghanistan. Bin Laden allegedly told him to "eliminate the infidels from the Arabian Peninsula."

From there he rose through the ranks. He was assigned in Aden to videotape the 1998 suicide bombing of the USS Cole, but he fell asleep.

Despite the lapse, the local leader, Nasser al-Wahishi, declared him the regional leader in Aden. He was also believed to have played a prominent role in al-Qaida's attack and capture last year of Zinjibar, the capital of Abyan province.

Government troops are trying to drive al-Qaida out of Zinjibar.

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Blind activist: What did he do to rile Beijing?

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Secretary of State Hillary Clinton said "progress had been made" on a deal over the future of Chen Guangcheng, the blind Chinese dissident at the center of an international firestorm. NBC's Ian Williams reports.

By NBC News Beijing

BEIJING ? Although blind human rights activist Chen Guangcheng has been cast into the international spotlight since his April 22 escape from house arrest and subsequent journey to the U.S. Embassy in Beijing, he still remains an unfamiliar name ??even to the majority of Chinese citizens, thanks to the country?s tight control of the press and social media.?

On Friday, the U.S. and China seemed to have forged the outlines of a tentative deal to end the diplomatic standoff that would let Chen travel to the U.S. with his family for a university fellowship. In the meantime, Chen?s fate still hangs in the balance.

So what exactly did he do to anger Chinese authorities so much in the first place? It all began with Chen?s foray into social activism nearly 16 years ago, when he began fighting against the Linyi government.?

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Challenging authority

Born on Nov. 12, 1971, Chen and grew-up in a small village called Dongshigu, near Linyi City in the eastern Shandong Province, approximately 400 miles from Beijing. He lost his sight after a severe fever when he was only a few months old.?

He enrolled in Qingdao High School for the Blind in 1994 and graduated in 1998. It was during this time that he had his first experience questioning authority. ?

Deal nears on China activist Chen as US offers college fellowship

In 1996 Chen traveled to Beijing to challenge the Linyi government?s illegal taxing of disabled people even though a 1991 law exempted them from taxes. He won the appeal.?

He kept fighting and in 1997 he irritated the local government again by appealing on behalf of his fellow villagers in a Beijing court to stop Linyi from breaking land laws.

Chen pursued these cases all without a formal law degree; he was a self-taught lawyer who had studied acupuncture and massage at the Nanjing University of Chinese Medicine from 1998 to 2001. His mother originally wanted him to become a masseur, the most common job for blind men in China, but he insisted on taking law classes on the side.

In September 2003, Chen sued the company that runs the Beijing subway system for making him buy subway tickets, despite the fact that the law said the subway should be free for disabled people. Chen again won the lawsuit.

Chinese crackdown on dissident's family and friends

Fighting abusive enforcement of the one-child policy
But what really brought Chen into the crosshairs of the Chinese government were his efforts to expose harsh illegal measures by local authorities in his hometown, Linyi, ?as they enforced China?s strict population control policy known as the ?one-child policy.??

Chen married Yuan Weijing in 2003 and their first son was born that year; in August 2005, they had a daughter. Some say the fact that they had two children, in defiance of China?s one-child policy, explains why Chen became interested in protesting family planning. ?

In 2005, the Linyi government started a campaign to "strictly enforce" the one-child policy by arresting and beating up women who broke the family planning law, forcing them to have abortions or sterilizations, heavily fining them and even arresting the relatives of those who had escaped to other cities. Chinese national laws prohibit such harsh acts.

Chen and Yuan investigated the cases and filed a class action lawsuit, while also revealing the brutality to the media.?The lawsuit was rejected, but through Chen?s work the brutality of Linyi officials was exposed and drew attention from both domestic and international press.?

(In particular, a Washington Post story in 2005, ?Who Controls the Family???first drew international attention to Chen?s crusade.?And apparently in thanks for that story, one of Chen?s first calls to the international media after leaving the U.S. embassy this week went to the Washington Post). ? ?

China censors 'Shawshank' as Clinton heads to Beijing amid dissident drama

Jail, then oppressive house arrest
After Chen refused negotiations with local officials to cease his activism, Chen was taken away by the police in March 2006. He was then officially arrested in June, and sentenced to four years and three months in prison for "destruction of property and disturbing public order." His trial was a controversial one because his lawyers were detained by Yinan police on the eve of the trial, leaving him defenseless in court.

During his jail sentence, in July 2008, his wife Yuan issued a public letter to Chinese President Hu Jintao, in which she said: "I hope the country?s leader can feel the insult and helplessness I have in my everyday life. I hope the leader will listen to a jailed blind man?s concern about the country?s future."?

Chen was released from prison in 2010, but then he and his wife were subjected to house arrest which included constant 24-hour video surveillance. In February 2011 he smuggled out a video showing his life under house arrest.?

?I was in a small prison, and now I am in a larger prison," Chen says to the camera in the hour-long video, which shows security agents peering over walls into the family?s home. ?

According to another video Chen released after his recent escape from house arrest, he estimated that authorities spent as much as 60 million yuan ($9.5 million) to keep him locked up.

True belief in the rule of law
During his one-and-half year long house detention, hundreds of people, including both Chinese and foreign journalists, lawyers, friends and human rights advocates, attempted to visit Chen but were all driven away, often violently by the thugs watching him day and night.?

In December 2011, Hollywood actor Christian Bale made an effort to see him along with a CNN crew and he was shoved away.?

Chen?s supporters say that ultimately his goal is to see that China lives up to the rule of law that already exists there.?

Blind dissident's case a 'hot potato' for US-China relations

"Chen Guangcheng is someone who really believes in rule of law, and he wants to put what?s written in the law into practice," said Zeng Jinyan, a long-time friend of Chen family and also a human rights activist. "While so many people who can see are still talking about securing personal safety, Chen, a blind man, is already in action."?

"People who know Chen say he is a Gandhi-esque figure and has a deep optimism that China will inevitably become a country ruled by law,? professor Susan L. Shirk, an expert on Chinese politics at the University of California in San Diego, told NBC News. ?He is not a dissident, agitating for a change in government ? he just wants China to enforce its own laws." ?

Many people attribute Chen?s ordeal to local government enforcement, arguing what they do is out of the ordinary, while many other believe it?s an order from the very top.?

"Evidently, local officials in Linyi concluded that Chen would somehow threaten local stability if he were free to move about and speak up. Beijing did not intervene even when it realized that the actions of the Linyi officials were creating a national and international embarrassment,? Kenneth Lieberthal, a China expert based in Beijing, told NBC News. ?The enormous reluctance by Beijing to intervene in these types of local decisions is the rule, not the exception."?

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Scientists aim to kill lung tumors

Public release date: 4-May-2012
[ | E-mail | Share ]

Contact: Dr. Rajkumar Savai
rajkumar.savai@innere.med.uni-giessen.de
49-603-270-5313
Max-Planck-Gesellschaft

Enzyme regulates the division of tumor cells and blood vessel growth in the cancer tissue

This release is available in German.

Lung cancer is the leading cause of cancer death throughout the world. Standard treatment methods do not usually result in long-term recovery. In addition to the proliferation of the tumour cells, the growth of blood vessels controls tumors development. The blood vessel growth is controlled by several signalling molecules. Scientists from the Max Planck Institute for Heart and Lung Research in Bad Nauheim and Justus Liebig University Giessen have discovered a molecule that plays a key role in this process. They succeeded in reducing tumour growth in their experiments by blocking the phosphodiesterase PDE4.

Lung cancer mainly affects smokers; however the disease can also be caused by contact with carcinogenic substances like asbestos. Chemotherapy or radiotherapy often prove insufficient in treating the disease. Hence, scientists are engaged in an intensive search for ways of halting the growth of lung tumours. The blood vessels that supply the tumour with nutrients offer a potential point of attack.

New blood vessels form to ensure an adequate supply of nutrients to the growing tumour. The growing tissue is immediately penetrated by blood vessels. The growth of the blood vessels is regulated by the tumour cells using a complex signal cascade, which is triggered initially by a low oxygen content (hypoxia) in the tumour tissue. "This state, which is known as hypoxia prompts the activation of around 100 genes in the tumour cells," explains Rajkumar Savai, research group leader at the Max Planck institute. "In addition to the growth of blood vessels, hypoxia also stimulates the proliferation of lung cancer cells." Three molecules play a particularly important role in this process. The activation of the genes at the beginning of the cascade is triggered by the transcription factor HIF and a messenger molecule, cAMP, is involved again at the end of the cascade. The researchers examined the third molecule that acts as a link between these two molecules in detail.

The molecule in question is a phosphodiesterase, PDE4. The scientists from Bad Nauheim and Giessen were able to demonstrate in their study that various sections of PDE4 have binding sites for HIF.

The researchers then tested the influence of a PDE4 blockade on the cells from ten different cell lines, which are characteristic of around 80 percent of lung cancers, in the laboratory. The rate of cell division in the cells treated with a PDE4 inhibitor was significantly lower and the HIF level also declined as a result.

The effect in the tumour bearing mice was particularly obvious. To observe this, the Max Planck researchers implanted a human tumour cell line under the skin of nude mice and treated the animals with the phosphodiesterase 4 inhibitor.

Tumour growth in these animals declined by around 50 percent. "Our microscopic analysis revealed that the blood vessel growth in the tumours of the mice that had been treated with the inhibitor was significantly reduced. We also observed indicators of decelerated cell division in the tumour cells. Overall, the tumour growth was strongly curbed."

Werner Seeger, Director of the MPI and Medical Director of the JLU University Hospital Giessen, reports: "We were able to show that PDE4 plays an important regulation function in cell division in lung tumours and in the development of blood vessels in cancer. Therefore, we hope that we have found a starting point for the development of a treatment here." In the view of tumour specialist Friedrich Grimminger, Chairman of the Department of Medical Oncology in Giessen, it may be possible in future to combine the inhibition of PDE4 with traditional radiotherapy or chemotherapy. In this way, the effect of the traditional treatment measures could be reinforced and patient prognoses may improve as a result. However, further laboratory studies are required before clinical tests can be carried out.

###

Original publication: S. Pullamsetti, G. Banat, A. Schmall, M. Szibor, D. Pomagruk, J. Hnze, E. Kolosionek, J. Wilhelm, T. Braun, F. Grimminger, W. Seeger, R. Schermuly, R. Savai: Phosphodiesterase-4 promotes proliferation and angiogenesis of lung cancer by crosstalk with HIF. Oncogene (2012). doi:10.1038/onc.2012.136

---

[ | E-mail | Share ]

?

AAAS and EurekAlert! are not responsible for the accuracy of news releases posted to EurekAlert! by contributing institutions or for the use of any information through the EurekAlert! system.

Public release date: 4-May-2012
[ | E-mail | Share ]

Contact: Dr. Rajkumar Savai
rajkumar.savai@innere.med.uni-giessen.de
49-603-270-5313
Max-Planck-Gesellschaft

Enzyme regulates the division of tumor cells and blood vessel growth in the cancer tissue

This release is available in German.

Lung cancer is the leading cause of cancer death throughout the world. Standard treatment methods do not usually result in long-term recovery. In addition to the proliferation of the tumour cells, the growth of blood vessels controls tumors development. The blood vessel growth is controlled by several signalling molecules. Scientists from the Max Planck Institute for Heart and Lung Research in Bad Nauheim and Justus Liebig University Giessen have discovered a molecule that plays a key role in this process. They succeeded in reducing tumour growth in their experiments by blocking the phosphodiesterase PDE4.

Lung cancer mainly affects smokers; however the disease can also be caused by contact with carcinogenic substances like asbestos. Chemotherapy or radiotherapy often prove insufficient in treating the disease. Hence, scientists are engaged in an intensive search for ways of halting the growth of lung tumours. The blood vessels that supply the tumour with nutrients offer a potential point of attack.

New blood vessels form to ensure an adequate supply of nutrients to the growing tumour. The growing tissue is immediately penetrated by blood vessels. The growth of the blood vessels is regulated by the tumour cells using a complex signal cascade, which is triggered initially by a low oxygen content (hypoxia) in the tumour tissue. "This state, which is known as hypoxia prompts the activation of around 100 genes in the tumour cells," explains Rajkumar Savai, research group leader at the Max Planck institute. "In addition to the growth of blood vessels, hypoxia also stimulates the proliferation of lung cancer cells." Three molecules play a particularly important role in this process. The activation of the genes at the beginning of the cascade is triggered by the transcription factor HIF and a messenger molecule, cAMP, is involved again at the end of the cascade. The researchers examined the third molecule that acts as a link between these two molecules in detail.

The molecule in question is a phosphodiesterase, PDE4. The scientists from Bad Nauheim and Giessen were able to demonstrate in their study that various sections of PDE4 have binding sites for HIF.

The researchers then tested the influence of a PDE4 blockade on the cells from ten different cell lines, which are characteristic of around 80 percent of lung cancers, in the laboratory. The rate of cell division in the cells treated with a PDE4 inhibitor was significantly lower and the HIF level also declined as a result.

The effect in the tumour bearing mice was particularly obvious. To observe this, the Max Planck researchers implanted a human tumour cell line under the skin of nude mice and treated the animals with the phosphodiesterase 4 inhibitor.

Tumour growth in these animals declined by around 50 percent. "Our microscopic analysis revealed that the blood vessel growth in the tumours of the mice that had been treated with the inhibitor was significantly reduced. We also observed indicators of decelerated cell division in the tumour cells. Overall, the tumour growth was strongly curbed."

Werner Seeger, Director of the MPI and Medical Director of the JLU University Hospital Giessen, reports: "We were able to show that PDE4 plays an important regulation function in cell division in lung tumours and in the development of blood vessels in cancer. Therefore, we hope that we have found a starting point for the development of a treatment here." In the view of tumour specialist Friedrich Grimminger, Chairman of the Department of Medical Oncology in Giessen, it may be possible in future to combine the inhibition of PDE4 with traditional radiotherapy or chemotherapy. In this way, the effect of the traditional treatment measures could be reinforced and patient prognoses may improve as a result. However, further laboratory studies are required before clinical tests can be carried out.

###

Original publication: S. Pullamsetti, G. Banat, A. Schmall, M. Szibor, D. Pomagruk, J. Hnze, E. Kolosionek, J. Wilhelm, T. Braun, F. Grimminger, W. Seeger, R. Schermuly, R. Savai: Phosphodiesterase-4 promotes proliferation and angiogenesis of lung cancer by crosstalk with HIF. Oncogene (2012). doi:10.1038/onc.2012.136

---

[ | E-mail | Share ]

?

AAAS and EurekAlert! are not responsible for the accuracy of news releases posted to EurekAlert! by contributing institutions or for the use of any information through the EurekAlert! system.

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Happy birthday, Adele! Singing sensation is 24

Lucy Nicholson / Reuters file

Adele holds her six Grammy Awards in Los Angeles on Feb. 12.

By Rachel Brethauer, E! Online

"Rumor Has It" someone's turning 24 today. Yes, the record-breaking, multi-platinum queen of breakup songs is having a birthday!

The supertalented singer and songwriter kicked off her career with a bang, receiving two Grammy awards in 2009 for her first album, 19.

It hasn't all been awards and accolades, though -- her blockbuster second album, 21, was all about heartbreak, and in 2011, Adele had to cancel a number of tour dates in order to undergo microsurgery on her vocal cords.

But she was back with a bang at the 2012 Grammys, giving an amazing performance and walking away with a record-tying six awards. And the megahits just keep on coming -- Ms. Adkins recently snagged 18 Billboard Award nominations!

PHOTOS: Fashion Spotlight: Adele

We can't wait to see what she does next. Perhaps a happy birthday this year will lead to a happy album next year? No matter what this songbird does, we're sure it'll be music to our ears.

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PFT: Hargove is key player in bounty case

Getty Images

With Saints linebacker Jonathan Vilma and Saints defensive end Will Smith claiming that they had no involvement in any type of bounty program, former Saints defensive end Anthony Hargrove becomes a key figure in the looming appeal process.

Originally interviewed by NFL Security when the league first investigated the claims of a bounty against Brett Favre in 2010, Hargrove denied the allegations.? In early March, Hargrove reiterated, in somewhat vague terms, his position that he had no involvement in a bounty system.

?I have made many mistakes in my life and have paid dearly for some of them, and the late hit and the comments were both mistakes, in my opinion,? Hargrove said, in reference to hitting Favre low and exclaiming ?Favre is out of the game!? Favre is done!? Favre is done!? thereafter.? ?But players all over the league do the same thing every Sunday, make late hits and say stupid things.? But I can say with absolute certainty that neither the late hit nor the comment have anything whatsoever to do with the issue being so hotly discussed in the media.?

On Wednesday, the NFL?s announcement of the various player suspensions mentioned that Hargrove has signed a ?declaration? (it?s like an affidavit, but it?s not signed in the presence of a notary) in which he ?established not only the existence of the program with the Saints, but also that he knew about and participated in it,? and that he ?told at least one player on another team that Vikings quarterback Brett Favre was a target of a large bounty.?

On Thursday, lawyer Mary Jo White described generally the contents of Hargrove?s declaration.? ?In it, he acknowledges the nature of the program and his participation in it,? White said.? ?And, which was really the thrust of the declaration, that he was told to lie about it and he did when he was asked about it in 2010 by the NFL investigators.?

White said Hargrove identified the person who told him to lie, but White declined to reveal the information.

And that highlights the primary disconnect with this process.? The NFL wants the media and, in turn, the public to have confidence in the investigation, but the NFL wants the media and, in turn, the public to simply take the NFL at its word.? At some point, there must be full disclosure of all relevant facts, if the NFL wants the media and, in turn, the public to approve the process.

This includes disclosing the full Hargrove declaration, along with all other evidence that proves the players who have been suspended did what they are accused of doing.? Given that the players who have been suspended are challenging their punishments and, at least as to Vilma and Smith, professing their innocence, far more is needed than mere summaries or conclusions.

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Limitless: The Brain-Doping Movie We All Know Was Inspired by Adderall [Video]

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